@dr.lucasluquetti

1 post audited · 3 claims analysed

Instagram profile

Science evidence grade

67%Moderate evidence grade

Based on 3 claims across 1 audit

SupportedOverstatedMisleadingNo Evidence

2

Supported

67%

0

Overstated

0%

0

Misleading

0%

1

No Evidence

33%

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Claim-level evidence grades — not a character judgment. Methodology · Right of reply · Leaderboards

What @dr.lucasluquetti claims actually are

We separate claims into three buckets: backed by evidence, factually incorrect, and grey — like animal-only findings sold as human fact (e.g. BPC-157 “fixes Achilles” from rat studies).

Evidence-based

67%

2 claims

Claims that align with published human or clinical evidence at the stated strength.

Ex: “Semaglutide can reduce body weight in adults with obesity” — supported by large RCTs.

Factually incorrect

0%

0 claims

Claims that conflict with the evidence, invent certainty, or omit critical safety/context in a misleading way.

Ex: “Peptides have no side effects” — contradicts known adverse-event profiles.

Grey / overstated

33%

1 claim

Plausible direction but wrong certainty — animal-only data sold as human fact, dose/effect overstated, or no adequate published support yet.

Ex: “BPC-157 fixes Achilles tears” — often rests on rodent tendon models, not proven human Achilles repair trials.

Evidence mix

Share of audited claims in each bucket

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BackedIncorrectGrey

Verdict detail

Grey splits into overstated (wrong certainty) vs no published support

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Claims over time

Stacked by bucket as audits land — plus the running evidence grade

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Gold line = running science evidence grade (Supported + ½ Overstated ÷ total claims).

Audit history(1 post)

Post thumbnail
Supported
Posted Nov 21, 2025·8 months ago·View source

O SS-31 acaba de ser liberado pela FDA. Um peptídeo mitocondrial que reduz estresse oxidativo, melhora a função celular e potencializa a performance metabólica. A ciência segue avançando e nós seguimos juntos.

SS-31 (elamipretide) has credible preclinical support for reducing oxidative stress and enhancing mitochondrial bioenergetic performance in cell culture and animal models. The oxidative stress reduction claim is well-documented across multiple independent studies. However, all available evidence is from in-vitro or animal research; no peer-reviewed human clinical trials confirming these outcomes in humans were found. The claim about 'improved cellular function' lacks specific literature. Creators should note that while the mechanistic direction is scientifically supported, human efficacy remains unproven.

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